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Akila Mayeda
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Regulation of alternative pre-mRNA splicing by hnRNP A1 and splicing factor SF2
A Mayeda, AR Krainer
Cell 68 (2), 365-375, 1992
9571992
Functional expression of cloned human splicing factor SF2: homology to RNA-binding proteins, U1 70K, and Drosophila splicing regulators
AR Krainer, A Mayeda, D Kozak, G Binns
Cell 66 (2), 383-394, 1991
6431991
Characterization of RNase R-digested cellular RNA source that consists of lariat and circular RNAs from pre-mRNA splicing
H Suzuki, Y Zuo, J Wang, MQ Zhang, A Malhotra, A Mayeda
Nucleic acids research 34 (8), e63-e63, 2006
6242006
Exon identity established through differential antagonism between exonic splicing silencer-bound hnRNP A1 and enhancer-bound SR proteins
J Zhu, A Mayeda, AR Krainer
Molecular cell 8 (6), 1351-1361, 2001
5482001
Regulated tissue-specific expression of antagonistic pre-mRNA splicing factors
A Hanamura, JF Cáceres, A Mayeda, BR Franza Jr, AR Krainer
Rna 4 (4), 430-444, 1998
4481998
Function of conserved domains of hnRNP A1 and other hnRNP A/B proteins.
A Mayeda, SH Munroe, JF Cáceres, AR Krainer
The EMBO journal 13 (22), 5483-5495, 1994
4201994
General splicing factor SF2/ASF promotes alternative splicing by binding to an exonic splicing enhancer.
Q Sun, A Mayeda, RK Hampson, AR Krainer, FM Rottman
Genes & development 7 (12b), 2598-2608, 1993
3771993
Identification and characterization of three members of the human SR family of pre‐mRNA splicing factors.
GR Screaton, JF Caceres, A Mayeda, MV Bell, M Plebanski, DG Jackson, ...
The EMBO journal 14 (17), 4336-4349, 1995
3651995
Modulation of exon skipping and inclusion by heterogeneous nuclear ribonucleoprotein A1 and pre-mRNA splicing factor SF2/ASF
A Mayeda, DM Helfman, AR Krainer
Molecular and cellular biology 13 (5), 2993-3001, 1993
3531993
hnRNP A/B proteins are required for inhibition of HIV-1 pre-mRNA splicing
M Caputi, A Mayeda, AR Krainer, AM Zahler
The EMBO journal 18 (14), 4060-4067, 1999
3501999
General splicing factors SF2 and SC35 have equivalent activities in vitro, and both affect alternative 5'and 3'splice site selection.
XD Fu, A Mayeda, T Maniatis, AR Krainer
Proceedings of the National Academy of Sciences 89 (23), 11224-11228, 1992
3311992
Selection of alternative 5′ splice sites: role of U1 snRNP and models for the antagonistic effects of SF2/ASF and hnRNP A1
IC Eperon, OV Makarova, A Mayeda, SH Munroe, JF Cáceres, ...
Molecular and cellular biology 20 (22), 8303-8318, 2000
2692000
Pathways for selection of 5′ splice sites by U1 snRNPs and SF2/ASF.
IC Eperon, DC Ireland, RA Smith, A Mayeda, AR Krainer
The EMBO journal 12 (9), 3607-3617, 1993
2611993
Identification of cis-and trans-acting factors involved in the localization of MALAT-1 noncoding RNA to nuclear speckles
R Miyagawa, K Tano, RIE Mizuno, YO Nakamura, K Ijiri, R Rakwal, ...
Rna 18 (4), 738-751, 2012
2472012
MALAT1 long non-coding RNA in cancer
R Yoshimoto, A Mayeda, M Yoshida, S Nakagawa
Biochimica et biophysica acta (BBA)-gene regulatory mechanisms 1859 (1), 192-199, 2016
2452016
Disruption of pre-mRNA splicing in vivo results in reorganization of splicing factors
RT O'Keefe, A Mayeda, CL Sadowski, AR Krainer, DL Spector
The Journal of cell biology 124 (3), 249-260, 1994
2191994
Substrate specificities of SR proteins in constitutive splicing are determined by their RNA recognition motifs and composite pre-mRNA exonic elements
A Mayeda, GR Screaton, SD Chandler, XD Fu, AR Krainer
Molecular and cellular Biology, 1999
2011999
Purification and characterization of human RNPS1: a general activator of pre-mRNA splicing
A Mayeda, J Badolato, R Kobayashi, MQ Zhang, EM Gardiner, AR Krainer
The EMBO journal 18 (16), 4560-4570, 1999
1911999
The subcellular localization of SF2/ASF is regulated by direct interaction with SR protein kinases (SRPKs)
J Koizumi, Y Okamoto, H Onogi, A Mayeda, AR Krainer, M Hagiwara
Journal of Biological Chemistry 274 (16), 11125-11131, 1999
1871999
Two members of a conserved family of nuclear phosphoproteins are involved in pre-mRNA splicing.
A Mayeda, AM Zahler, AR Krainer, MB Roth
Proceedings of the National Academy of Sciences 89 (4), 1301-1304, 1992
1771992
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