George Hajishengallis
George Hajishengallis
Thomas W. Evans Centennial Professor, University of Pennsylvania
Verified email at - Homepage
Cited by
Cited by
Complement: a key system for immune surveillance and homeostasis
D Ricklin, G Hajishengallis, K Yang, JD Lambris
Nature immunology 11 (9), 785-797, 2010
Periodontitis: from microbial immune subversion to systemic inflammation
G Hajishengallis
Nature reviews immunology 15 (1), 30-44, 2015
The keystone-pathogen hypothesis
G Hajishengallis, RP Darveau, MA Curtis
Nature Reviews Microbiology 10 (10), 717-725, 2012
The oral microbiota: dynamic communities and host interactions
RJ Lamont, H Koo, G Hajishengallis
Nature reviews microbiology 16 (12), 745-759, 2018
Beyond the red complex and into more complexity: the polymicrobial synergy and dysbiosis (PSD) model of periodontal disease etiology
G Hajishengallis, RJ Lamont
Molecular oral microbiology 27 (6), 409-419, 2012
Low-abundance biofilm species orchestrates inflammatory periodontal disease through the commensal microbiota and complement
G Hajishengallis, S Liang, MA Payne, A Hashim, R Jotwani, MA Eskan, ...
Cell host & microbe 10 (5), 497-506, 2011
Immunomicrobial pathogenesis of periodontitis: keystones, pathobionts, and host response
G Hajishengallis
Trends in immunology 35 (1), 3-11, 2014
Modulation of myelopoiesis progenitors is an integral component of trained immunity
I Mitroulis, K Ruppova, B Wang, LS Chen, M Grzybek, T Grinenko, ...
Cell 172 (1), 147-161. e12, 2018
Local and systemic mechanisms linking periodontal disease and inflammatory comorbidities
G Hajishengallis, T Chavakis
Nature Reviews Immunology 21 (7), 426-440, 2021
TLR-signaling networks: an integration of adaptor molecules, kinases, and cross-talk
J Brown, H Wang, GN Hajishengallis, M Martin
Journal of dental research 90 (4), 417-427, 2011
Polymicrobial synergy and dysbiosis in inflammatory disease
RJ Lamont, G Hajishengallis
Trends in molecular medicine 21 (3), 172-183, 2015
Porphyromonas gingivalis as a Potential Community Activist for Disease
RP Darveau, G Hajishengallis, MA Curtis
Journal of dental research 91 (9), 816-820, 2012
The use of rodent models to investigate host–bacteria interactions related to periodontal diseases
DT Graves, D Fine, YTA Teng, TE Van Dyke, G Hajishengallis
Journal of clinical periodontology 35 (2), 89-105, 2008
The leukocyte integrin antagonist Del-1 inhibits IL-17-mediated inflammatory bone loss
MA Eskan, R Jotwani, T Abe, J Chmelar, JH Lim, S Liang, PA Ciero, ...
Nature immunology 13 (5), 465-473, 2012
Optimization of the ligature-induced periodontitis model in mice
T Abe, G Hajishengallis
Journal of immunological methods 394 (1-2), 49-54, 2013
A new inflammatory cytokine on the block: re-thinking periodontal disease and the Th1/Th2 paradigm in the context of Th17 cells and IL-17
SL Gaffen, G Hajishengallis
Journal of dental research 87 (9), 817-828, 2008
Novel mechanisms and functions of complement
G Hajishengallis, ES Reis, DC Mastellos, D Ricklin, JD Lambris
Nature immunology 18 (12), 1288-1298, 2017
Basic biology and role of interleukin‐17 in immunity and inflammation
C Zenobia, G Hajishengallis
Periodontology 2000 69 (1), 142-159, 2015
The inflammophilic character of the periodontitis‐associated microbiota
G Hajishengallis
Molecular oral microbiology 29 (6), 248-257, 2014
Porphyromonas gingivalis manipulates complement and TLR signaling to uncouple bacterial clearance from inflammation and promote dysbiosis
T Maekawa, JL Krauss, T Abe, R Jotwani, M Triantafilou, K Triantafilou, ...
Cell host & microbe 15 (6), 768-778, 2014
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